Lupus erythematosus is a disease, which imitates others so well that it can easily be misdiagnosed. Its symptoms imitate or overlap with other health problems.
However, despite its complex and even dual characteristics, Lupus, having been correctly diagnosed, is successfully treated only with a causal approach at a cellular level.
What is Systemic Lupus Erythematosus?
Lupus is a chronic autoimmune disease that causes systemic inflammation in multiple systems of the human body. Lupus can “attack” joints, skin, kidneys, blood cells, heart, lungs and brain, causing disabilities and sometimes fatal reactions. The most common form of lupus is known as systemic lupus erythematosus (SLE). However, there are rarer forms of the disease: discoid lupus erythematosus (DLE), subacute cutaneous lupus erythematosus (SCLE), drug-induced lupus, and neonatal lupus erythematosus.
The symptoms of lupus erythematosus.
Symptoms can vary and mimic other situations. However, the most common symptoms include:
- Butterfly type rash
- Sensitivity to the sun, which often leads to a rash or skin lesions.
- Fatigue and fever
- Joint pain, stiffness and swelling
- Raynaud’s syndrome (fingers turn white, then blue in response to cold or stress)
- Dry eyes
- Breathing difficulty
- Chest pain
- Memory loss and confusion
A butterfly rash or wolf mask is a warning sign of Lupus – although it does not develop in everyone who has the disease. It is a rash that covers the cheeks and nose in a butterfly pattern.
What causes the disease?
There are as many predisposing factors for this disease, such as Metabolic disorders (not enough acetyl coenzyme A), hormonal imbalance, chronic infections, medication, nutritional deficiencies and toxin overload.
It is well known that predisposing factors can play different roles in different people.
The causes of lupus are complex and varied, but all patients with lupus have three common problems that are characteristic of anyone else suffering from Autoimmune Disease.
These three problems so as converge to express their illness.
At first, genetic activity that creates a predisposition in the body. Secondly, damage to the gut from drugs, toxins, infections, food or stress. Third, exposure to environmental pollution: contamination, sensitivity to food, toxic metals and / or other environmental chemicals.
In terms of genetic predisposition, people with lupus often have a family history of an autoimmune disease, not necessarily lupus. However, about two dozen genes have been discovered that contribute to the occurrence of Lupus.
When the gut microbiome is dysregulated, issues such as the leaky gut syndrome may arise and subsequently lead to chronic inflammation. This inflammation does not necessarily remain local. It can spread to other organs and parts of the body. Genetically modified or over-processed foods are known to affect the lining of the gut, destroying healthy gut bacteria.
Finally, environmental factors are everywhere.
More than 90,000 chemical substances have been released on the planet since 1900. Few have been tested for safety. Some of the many sources of toxins include cleaning products and air fresheners, mercury, from carbon from combustion power plants and from amalgam filling in our teeth, from food.
Many doctors and scientists are convinced that the dramatic increase in the disease over the last two decades, as well as other Autoimmune diseases, is due to an overwhelming “poisoning “ of our body by numerous toxins that are combined with an inadequate diet of micro and macronutrients.
The complications of lupus erythematosus can be serious
Kidney failure is one of the leading causes of death among people with lupus. However, other possible complications include:
- Pericarditis (inflammation of the heart membrane)
- Cardiovascular disease
- Heart attack
- Inflammation of the chest wall cavity
- Blood clotting disorders
- Vasculitis (inflammation of the blood vessels)
- Behavior changes
- Memory problems
- Susceptibility to infection
- Increased risk of cancer
- Complications in pregnancy
- Predisposition to other autoimmune diseases
But before we talk about the possible treatments for lupus, let’s look at the classic treatment of Autoimmune diseases.
In this classic therapeutic approach, the treatment of Lupus and other Autoimmune Diseases follows two paths:
(1) based on symptoms
(2) with “aggressive” disease control.
The first does not address the overall progression of the disease. While the second involves the use of drugs with many side effects, without any substantial causal effect.
Etiological therapeutic approaches
In contrast, an etiological therapeutic approach can yield spectacular results in Lupus erythematosus.
This means that once we record a person’s overall state of health, lifestyle, diet and how the environment has contributed to the disease, we can gradually restore cellular balance.
Autoimmune Diseases, Curing the Causes
The key to treatments lies deep within the cell and its mechanisms.
Special Molecular and Genetic Tests can detect the true causes and mechanisms of the disease.
The biochemical diversion that led to the onset of the Autoimmune Disease is gradually restored, by administering strictly personalized therapeutic protocols based on clinical algorithms.
The overall clinical image and health levels are improving overall. Patients who additionally follow the instructions given, after the end of treatment, reduce the chances of a disease relapse to a minimum.
These treatments have been used successfully for at least the last twenty years and do not interfere with any other medications.
1. “Handout on Health: Systemic Lupus Erythematosus”. www.niams.nih.gov. February 2015. Archived from the original on 17 June 2016. Retrieved 12 June 2016.
2. Davis, Laurie S.; Reimold, Andreas M. (April 2017). “Research and therapeutics—traditional and emerging therapies in systemic lupus erythematosus”. Rheumatology. 56 (suppl_1): i100–i113. doi:10.1093/rheumatology/kew417. PMC 5850311. PMID 28375452.
3. Gladman, Dafna (10 September 2015). “Overview of the clinical manifestations of systemic lupus erythematosus in adults”. UpToDate. Archived from the original on 19 April 2017. Retrieved 18 April 2017.
4. Singh, Jasvinder A.; Hossain, Alomgir; Kotb, Ahmed; Oliveira, Ana; Mudano, Amy S.; Grossman, Jennifer; Winthrop, Kevin; Wells, George A. (2016). “Treatments for Lupus Nephritis: A Systematic Review and Network Metaanalysis”. The Journal of Rheumatology. 43 (10): 1801–1815. doi:10.3899/jrheum.160041. ISSN 0315-162X. PMID 27585688. S2CID 19621372.
5. Kasama, T; Maeoka, A; Oguro, N (2016). “Clinical Features of Neuropsychiatric Syndromes in Systemic Lupus Erythematosus and Other Connective Tissue Diseases”. Clinical Medicine Insights. Arthritis and Musculoskeletal Disorders. 9: 1–8. doi:10.4137/CMAMD.S37477. PMC 4718090. PMID 26819561.
6. Ünlü, O; Zuily, S; Erkan, D (2016). “The clinical significance of antiphospholipid antibodies in systemic lupus erythematosus”. European Journal of Rheumatology. 3 (2): 75–84. doi:10.5152/eurjrheum.2015.0085. PMC 5042235. PMID 27708976.
7. Singh, RR; Yen, EY (September 2018). “SLE mortality remains disproportionately high, despite improvements over the last decade”. Lupus. 27 (10): 1577–1581. doi:10.1177/0961203318786436. PMC 6082727. PMID 30016928.
8. Lupus Foundation of America. “What is the history of lupus?”. Archived from the original on 4 November 2014. Retrieved 11 October 2014.